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Critical Role of Viral Protein Hexon in Hypervirulent Fowl Adenovirus Serotype-4-Induced Autophagy by Interaction with BAG3 and Promotion of Viral Replication in LMH Cells

Wei Li, Guangju You, Areayi Haiyilati, Hongnuan Wang, Huixuan Jiao, Yongqiang Wang, Li Gao, Hong Cao, Xiaoqi Li, Shijun J. Zheng

2023Journal of Virology18 citationsDOIOpen Access PDF

Abstract

HHS, mainly caused by HPFAdV-4, has caused large economic losses to the stakeholders in recent years. Infection of leghorn male hepatocellular (LMH) cells by HPFAdV-4 induced complete autophagy that is essential for HPFAdV-4 replication. By a screening strategy, the viral protein hexon was found responsible for virus-induced autophagy in cells. Importantly, hexon was identified as a factor promoting viral replication by interaction with BAG3, an initiator of host cell autophagy. These findings will help us to better understand the host response to HPFAdV-4 infection, providing a novel insight into the pathogenesis of HPFAdV-4 infection.

Topics & Concepts

BiologyVirologyViral replicationAutophagyBAG3Viral proteinReplication (statistics)SerotypeVirusGeneticsApoptosisVirus-based gene therapy researchViral gastroenteritis research and epidemiologyCytomegalovirus and herpesvirus research
Critical Role of Viral Protein Hexon in Hypervirulent Fowl Adenovirus Serotype-4-Induced Autophagy by Interaction with BAG3 and Promotion of Viral Replication in LMH Cells | Litcius