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EphrinB2 clustering by Nipah virus G is required to activate and trap F intermediates at supported lipid bilayer–cell interfaces

Joyce J. W. Wong, Zhongwen Chen, Jean K. Chung, Jay T. Groves, Theodore S. Jardetzky

2021Science Advances35 citationsDOIOpen Access PDF

Abstract

Paramyxovirus membrane fusion requires an attachment protein that binds to a host cell receptor and a fusion protein that merges the viral and host membranes. For Nipah virus (NiV), the G attachment protein binds ephrinB2/B3 receptors and activates F-mediated fusion. To visualize dynamic events of these proteins at the membrane interface, we reconstituted NiV fusion activation by overlaying F- and G-expressing cells onto ephrinB2-functionalized supported lipid bilayers and used TIRF microscopy to follow F, G, and ephrinB2. We found that G and ephrinB2 form clusters and that oligomerization of ephrinB2 is necessary for F activation. Single-molecule tracking of F particles revealed accumulation of an immobilized intermediate upon activation. We found no evidence for stable F-G protein complexes before or after activation. These observations lead to a revised model for NiV fusion activation and provide a foundation for investigating other multicomponent viral fusion systems.

Topics & Concepts

GlycoproteinLipid bilayer fusionTrap (plumbing)Viral entryVirusLipid bilayerChemistryBilayerCell biologyVirologyBiophysicsBiologyBiochemistryPhysicsViral replicationMembraneMeteorologyVirology and Viral DiseasesPlant Virus Research StudiesBacteriophages and microbial interactions
EphrinB2 clustering by Nipah virus G is required to activate and trap F intermediates at supported lipid bilayer–cell interfaces | Litcius