Litcius/Paper detail

Increased double-strand breaks in aged mouse male germ cells may result from changed expression of the genes essential for homologous recombination or nonhomologous end joining repair

Günel Talibova, Yesim Bilmez, Saffet Öztürk

2022Histochemistry and Cell Biology11 citationsDOI

Topics & Concepts

DNA repair protein XRCC4Ku80RAD51Non-homologous end joiningBiologyDNA repairHomologous recombinationSertoli cellGerm cellDNA damageSpermatogenesisCell biologyGeneAndrologyGeneticsDNADNA mismatch repairEndocrinologyDNA-binding proteinMedicineTranscription factorDNA Repair MechanismsReproductive Biology and FertilityCRISPR and Genetic Engineering
Increased double-strand breaks in aged mouse male germ cells may result from changed expression of the genes essential for homologous recombination or nonhomologous end joining repair | Litcius