Prolonged hypoxia alleviates prolyl hydroxylation-mediated suppression of RIPK1 to promote necroptosis and inflammation
Tao Zhang, Daichao Xu, Jianping Liu, Min Wang, Li‐Juan Duan, Min Liu, Huyan Meng, Yuan Zhuang, Huibing Wang, Yingnan Wang, Mingming Lv, Zhengyi Zhang, Jia Hu, Linyu Shi, Rui Guo, Xingxing Xie, Hui Liu, Emily C. Erickson, Yaru Wang, Wenyu Yu, Fabin Dang, Dongxian Guan, Cong Jiang, Xiaoming Dai, Hiroyuki Inuzuka, Peiqiang Yan, Jingchao Wang, Mrigya Babuta, Gewei Lian, Zhenbo Tu, Ji Miao, Gyöngyi Szabó, Guo‐Hua Fong, Antoine E. Karnoub, Yu-Ru Lee, Lifeng Pan, William G. Kaelin, Junying Yuan, Wenyi Wei
Topics & Concepts
NecroptosisRIPK1Cell biologyProgrammed cell deathInflammationHydroxylationChemistryHypoxia (environmental)Signal transductionApoptosisBiologyCancer researchBiochemistryImmunologyEnzymeOxygenOrganic chemistryCancer, Hypoxia, and MetabolismMitochondrial Function and PathologyCell death mechanisms and regulation