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Replication Kinetics, Cell Tropism, and Associated Immune Responses in SARS-CoV-2- and H5N1 Virus-Infected Human Induced Pluripotent Stem Cell-Derived Neural Models

Lisa Bauer, Bas Lendemeijer, Lonneke Leijten, Carmen W.E. Embregts, Barry Rockx, Steven A. Kushner, Femke M.S. de Vrij, Debby van Riel

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Abstract

Infections with the recently emerged severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) are often associated with neurological complications. Evidence suggests that SARS-CoV-2 enters the brain via the olfactory nerve; however, SARS-CoV-2 is only rarely detected in the central nervous system of COVID-19 patients. Here, we show that SARS-CoV-2 is able to infect neurons of human iPSC neural cultures but that this infection is abortive and does not result in virus spread to other cells. However, infection of neural cultures did result in the production of type III interferon and IL-8. This study suggests that SARS-CoV-2 might enter the CNS and infect individual neurons, triggering local immune responses that could contribute to the pathogenesis of SARS-CoV-2-associated CNS disease.

Topics & Concepts

BiologyTropismVirologyCoronavirusVirusImmunologyViral replicationInduced pluripotent stem cellImmune systemTissue tropismInfluenza A virusMedicinePathologyEmbryonic stem cellCoronavirus disease 2019 (COVID-19)BiochemistryGeneInfectious disease (medical specialty)DiseaseLong-Term Effects of COVID-19SARS-CoV-2 and COVID-19 ResearchInfectious Encephalopathies and Encephalitis
Replication Kinetics, Cell Tropism, and Associated Immune Responses in SARS-CoV-2- and H5N1 Virus-Infected Human Induced Pluripotent Stem Cell-Derived Neural Models | Litcius