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The V654A second-site KIT mutation increases tumor oncogenesis and STAT activation in a mouse model of gastrointestinal stromal tumor

Jennifer Q. Zhang, Benedikt Bosbach, Jennifer K. Loo, Gerardo A. Vitiello, Shan Zeng, Adrian M. Seifert, Benjamin D. Medina, Nesteene J. Param, Joanna H. Maltbaek, Ferdinand Rossi, Cristina R. Antonescu, Peter Besmer, Ronald P. DeMatteo

2020Oncogene14 citationsDOIOpen Access PDF

Topics & Concepts

Cancer researchBiologySunitinibImatinibProto-Oncogene Proteins c-kitInterstitial cell of CajalMutationGiSTStromal tumorStromal cellCarcinogenesisTyrosine-kinase inhibitorCD117Tyrosine kinaseCancerImmunologyCD34Signal transductionGeneticsStem cell factorHaematopoiesisImmunohistochemistryGeneStem cellMyeloid leukemiaGastrointestinal Tumor Research and TreatmentGastric Cancer Management and OutcomesGastrointestinal disorders and treatments
The V654A second-site KIT mutation increases tumor oncogenesis and STAT activation in a mouse model of gastrointestinal stromal tumor | Litcius