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Cannabinoid Receptor 1/miR-30b-5p Axis Governs Macrophage NLRP3 Expression and Inflammasome Activation in Liver Inflammatory Disease

Le Yang, Lei Tian, Zhi Zhang, Xuan Zhou, Xiaofang Ji, Fuquan Liu, Chengbin Dong, Lei Hou, Xinhao Zhao, Na Chang, Lin Yang, Liying Li

2020Molecular Therapy — Nucleic Acids34 citationsDOIOpen Access PDF

Abstract

- and MCDHF-treated mice. MicroRNA-30b-5p (miR-30b-5p), screened by the intersection of bioinformatics databases and downregulated miRNAs in injured liver, negatively correlated with NLRP3 in mouse and human liver. miR-30b-5p was involved in CB1-mediated activation of NLRP3 inflammasome in macrophages by directly targeting NLRP3. Importantly, administration of miR-30b-5p agomir targeted NLRP3 and attenuated liver inflammation in the injured liver. Altogether, CB1/miR-30b-5p axis modulates NLRP3 expression and NLPR3 inflammasome activation in macrophages during liver inflammation, which provides a potential target for liver disease.

Topics & Concepts

InflammasomePyrin domainCannabinoid receptorInflammationLiver X receptorLiver injuryNodCannabinoidChemistryReceptorImmunologyAgonistMedicinePharmacologyBiochemistryNuclear receptorTranscription factorGeneInflammasome and immune disordersHeme Oxygenase-1 and Carbon MonoxideLiver Disease Diagnosis and Treatment