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P2RY2-AKT activation is a therapeutically actionable consequence of XPO1 inhibition in acute myeloid leukemia

Kevin Lin, Justine C. Rutter, Abigail Xie, Shane T. Killarney, Camille Vaganay, Chaı̈ma Benaksas, Frank Ling, Gaetano Sodaro, Paul-Arthur Meslin, Christopher F. Bassil, Nina Fenouille, Jacob P. Hoj, Rachel Washart, Hazel X. Ang, Christian Cerda-Smith, Paul Chaintreuil, Arnaud Jacquel, Patrick Auberger, Antoine Forget, Raphaël Itzykson, Min Lu, Jiaxing Lin, Mariaelena Pierobon, Zhecheng Sheng, Xinghai Li, Ashutosh Chilkoti, Kouros Owzar, David A. Rizzieri, Timothy S. Pardee, Lina Benajiba, Emanuel F. Petricoin, Alexandre Puissant, Kris C. Wood

2022Nature Cancer42 citationsDOIOpen Access PDF

Topics & Concepts

Protein kinase BMyeloid leukemiaPI3K/AKT/mTOR pathwayCancer researchDownregulation and upregulationLeukemiaSignal transductionPharmacologyBiologyImmunologyCell biologyGeneGeneticsAcute Myeloid Leukemia ResearchNuclear Structure and FunctionProtein Degradation and Inhibitors
P2RY2-AKT activation is a therapeutically actionable consequence of XPO1 inhibition in acute myeloid leukemia | Litcius