Litcius/Paper detail

PLXND1/SEMA3E Promotes Epithelial–Mesenchymal Transition Partly via the PI3K/AKT-Signaling Pathway and Induces Heterogenity in Colorectal Cancer

Kiyotaka Hagihara, Naotsugu Haraguchi, Junichi Nishimura, Asuka Yasueda, Shiki Fujino, Takayuki Ogino, Hidekazu Takahashi, Norikatsu Miyoshi, Mamoru Uemura, Chu Matsuda, Tsunekazu Mizushima, Hirofumi Yamamoto, Masaki Mori, Yuichiro� Doki, Hidetoshi Eguchi

2022Annals of Surgical Oncology17 citationsDOIOpen Access PDF

Topics & Concepts

Epithelial–mesenchymal transitionFurinGene knockdownCancer researchCell migrationMetastasisPI3K/AKT/mTOR pathwayWnt signaling pathwaySMADPlexinProtein kinase BColorectal cancerMedicineCellTransforming growth factorCell biologySignal transductionChemistryCancerInternal medicineApoptosisBiologyReceptorBiochemistryEnzymeSemaphorinAxon Guidance and Neuronal SignalingHedgehog Signaling Pathway StudiesCancer Cells and Metastasis
PLXND1/SEMA3E Promotes Epithelial–Mesenchymal Transition Partly via the PI3K/AKT-Signaling Pathway and Induces Heterogenity in Colorectal Cancer | Litcius