Cognitive Deficiencies pathophysiology are mainly an unknown area. Curing the neurological conditions could be an objective for gene therapy
Moataz Dowaidar
Abstract
Currently, it appears that the pathophysiology of Cognitive Deficiency (CD) is mostly uncharted territory. In this study, we used both clinical research and mouse models to identify the reported Systemic lupus erythematosus (SLE) symptoms and to explain how tissue damage occurs in SLE, which helps us develop optimal therapies for CD. The primary goal of treatment is to cure the neurological problem, rather than repair the immunological issues. It should be noted that CD develops even in the absence of flares, indicating that treating it would need much more intensive immunosuppression with more damage. For further study on CD, it will be necessary to collaborate amongst neurologists, rheumatologists, and immunologists. This work would succeed by advancing the ability to unveil or hide the fundamental mechanism of CD.