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Extracellular vesicles derived from Krüppel-Like Factor 2-overexpressing endothelial cells attenuate myocardial ischemia-reperfusion injury by preventing Ly6C<sup>high</sup> monocyte recruitment

Shuaihua Qiao, Wen‐Feng Zhang, Yong Yin, Zhonghai Wei, Fu Chen, Jinxuan Zhao, Xuan Sun, Dan Mu, Jun Xie, Biao Xu

2020Theranostics49 citationsDOIOpen Access PDF

Abstract

monocyte recruitment. Thus, KLF2-EVs could be a potential therapeutic agent for myocardial I/R injury.

Topics & Concepts

Extracellular vesiclesExtracellularIschemiaReperfusion injuryMonocyteMyocardial ischemiaCell biologyChemistryMedicineCardiologyCancer researchPharmacologyInternal medicineBiologyExtracellular vesicles in diseaseMicroRNA in disease regulationCircular RNAs in diseases
Extracellular vesicles derived from Krüppel-Like Factor 2-overexpressing endothelial cells attenuate myocardial ischemia-reperfusion injury by preventing Ly6C<sup>high</sup> monocyte recruitment | Litcius