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Female-specific synaptic dysfunction and cognitive impairment in a mouse model of <i>PCDH19</i> disorder

Naosuke Hoshina, Erin Johnson‐Venkatesh, Miyuki Hoshina, Hisashi Umemori

2021Science73 citationsDOIOpen Access PDF

Abstract

More is not always better Mutations in X-linked genes usually affect male individuals more than females, but the opposite characterizes the Protocadherin-19 gene ( PCDH19 ) on the X chromosome. Mutations in the PCDH19 cell-adhesion molecule cause cognitive impairment, affecting females more than males. Hoshina et al. studied mice with PCDH19 mutations, showing that a mismatch between PCDH19 and another cell-adhesion molecule causes trouble when mossy fibers of the brain are forming synapses (see the Perspective by Shohayeb and Cooper). In the heterozygous setting, mutant PCDG19 sequesters the partner cell-adhesion molecule into dysfunctional complexes. In the hemizygous setting, as in males, enough of that partner cell-adhesion molecule roams free to make functional interactions. Science , this issue p. eaaz3893 ; see also p. 235

Topics & Concepts

ProtocadherinMossy fiber (hippocampus)SynapsePhenotypeNeuroscienceHippocampal formationBiologyCadherinGeneGeneticsCellDentate gyrusWnt/β-catenin signaling in development and cancerNeuroscience and Neuropharmacology ResearchAlzheimer's disease research and treatments
Female-specific synaptic dysfunction and cognitive impairment in a mouse model of <i>PCDH19</i> disorder | Litcius