CC16 Deficiency in the Context of Early-Life <i>Mycoplasma pneumoniae</i> Infection Results in Augmented Airway Responses in Adult Mice
Natalie Iannuzo, Michael Insel, Craig Marshall, William P. Pederson, Kenneth J. Addison, Francesca Polverino, Stefano Guerra, Julie G. Ledford
Abstract
mouse tracheal epithelial cells (MTECs) were infected with Mp, increased Mp colonization and collagen gene expression were also detected compared to WT cells, suggesting that CC16 plays a protective role during Mp infection, in part through epithelial-driven host defense mechanisms.
Topics & Concepts
BiologyContext (archaeology)ImmunologyLungAirwayImmunityImmune systemSecretionRespiratory systemMycoplasma pneumoniaeRatónProinflammatory cytokineMycoplasmaCellEndocrinologyClearanceRespiratory tractInterleukin 6EosinophilChronic infectionRespiratory infectionInternal medicineNeonatal Respiratory Health ResearchChemokine receptors and signalingCystic Fibrosis Research Advances