Age-associated B cells contribute to the pathogenesis of rheumatoid arthritis by inducing activation of fibroblast-like synoviocytes via TNF-α-mediated ERK1/2 and JAK-STAT1 pathways
Yi Qin, Ming-Long Cai, Hui-Zhi Jin, Wei Huang, Zhu Chen, Aline Bözec, Jingang Huang, Zhu Chen
Topics & Concepts
PathogenesisMedicineImmunologyDownregulation and upregulationSynovial fluidArthritisFibroblastTumor necrosis factor alphaRheumatoid arthritisFlow cytometryCancer researchBiologyCell culturePathologyOsteoarthritisGeneGeneticsAlternative medicineBiochemistryRheumatoid Arthritis Research and TherapiesCholesterol and Lipid MetabolismAtherosclerosis and Cardiovascular Diseases