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Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis

Hyun Jik Lee, Young Hyun Jung, Gee Euhn Choi, Jun Sung Kim, Chang Woo Chae, Jae Ryong Lim, Seo Yihl Kim, Jee Hyeon Yoon, Ji Hyeon Cho, Sei‐Jung Lee, Ho Jae Han

2020Cell Death and Differentiation179 citationsDOIOpen Access PDF

Topics & Concepts

NeurodegenerationMitochondrionCell biologyChemistryEndoplasmic reticulumNeuroprotectionBiochemistryBiologyPharmacologyInternal medicineMedicineDiseasePomegranate: compositions and health benefitsParkinson's Disease Mechanisms and TreatmentsGinkgo biloba and Cashew Applications
Urolithin A suppresses high glucose-induced neuronal amyloidogenesis by modulating TGM2-dependent ER-mitochondria contacts and calcium homeostasis | Litcius