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Polydatin protects SH-SY5Y in models of Parkinson's disease by promoting Atg5-mediated but parkin-independent autophagy

Hua Bai, Yaqi Ding, Xin Li, Deqin Kong, Chenqi Xin, Xuekang Yang, Chengwu Zhang, Zi‐Qiang Rong, Chuanhao Yao, Shenci Lu, Lei Ji, Lin Li, Wei Huang

2020Neurochemistry International38 citationsDOI

Topics & Concepts

ParkinATG5AutophagyPINK1MFN2BiologyProgrammed cell deathOxidative stressNeuroprotectionSubstantia nigraCell biologyRotenonemitochondrial fusionMitochondrionParkinson's diseaseMitochondrial DNADopaminergicMitophagyPharmacologyApoptosisMedicineGeneticsBiochemistryEndocrinologyInternal medicineDopamineDiseaseGeneAutophagy in Disease and TherapyParkinson's Disease Mechanisms and TreatmentsSirtuins and Resveratrol in Medicine
Polydatin protects SH-SY5Y in models of Parkinson's disease by promoting Atg5-mediated but parkin-independent autophagy | Litcius