The interaction of oxidative stress with MAPK, PI3/AKT, NF-κB, and DNA damage kinases influences the fate of γ-radiation-induced bystander cells
Sharmi Mukherjee, Anindita Dutta, Anindita Chakraborty
Topics & Concepts
Bystander effectOxidative stressProtein kinase BSuperoxide dismutaseChemistryDNA damageCell biologyPI3K/AKT/mTOR pathwayReactive oxygen speciesKinaseMolecular biologySignal transductionCancer researchBiologyImmunologyBiochemistryDNAEffects of Radiation ExposureGenomics, phytochemicals, and oxidative stressCell death mechanisms and regulation