The Akt/mTOR and MNK/eIF4E pathways rewire the prostate cancer translatome to secrete HGF, SPP1 and BGN and recruit suppressive myeloid cells
Daniela Brina, Adele Ponzoni, Martina Troiani, Bianca Calì, Emiliano Pasquini, Giuseppe Attanasio, Simone Mosole, Michela Mirenda, Mariantonietta D’Ambrosio, Manuel Colucci, Ilaria Guccini, Ajinkya Revandkar, Abdullah Alajati, Toma Tebaldi, Deborah Donzel, Fabio Lauria, Nahjme Parhizgari, Aurora Valdata, Martino Maddalena, Arianna Calcinotto, Marco Bolis, Andrea Rinaldi, Simon T. Barry, Jan H. Rüschoff, Marianna Sabbadin, Semini Sumanasuriya, Mateus Crespo, Adam Sharp, Wei Yuan, Mathew Grinu, Alexandra Boyle, Cynthia L. Miller, Lloyd C. Trotman, Nicolas Delaleu, Matteo Fassan, Holger Moch, Gabriella Viero, Johann S. de Bono, Andrea Alimonti
Topics & Concepts
Prostate cancerPI3K/AKT/mTOR pathwayCancer researchEIF4EImmunotherapyProstateCancerTumor microenvironmentProtein kinase BImmune systemPTENMedicineTranslation (biology)BiologyImmunologySignal transductionInternal medicineMessenger RNAGeneCell biologyBiochemistryImmune cells in cancerImmune Cell Function and InteractionEpigenetics and DNA Methylation