Cytoplasmic TDP-43 accumulation drives changes in C-bouton number and size in a mouse model of sporadic Amyotrophic Lateral Sclerosis
Anna Normann Bak, Svetlana Djukic, Marion Kadlecová, Thomas Hartig Braunstein, Dennis Bo Jensen, Claire Francesca Meehan
Topics & Concepts
Amyotrophic lateral sclerosisBiologySOD1NeuroscienceSomaGenetically modified mouseMotor neuronCytoplasmPopulationNeurodegenerationTransgeneAnatomyCell biologyPathologySpinal cordDiseaseGeneticsMedicineGeneEnvironmental healthAmyotrophic Lateral Sclerosis ResearchNeurogenetic and Muscular Disorders ResearchPrion Diseases and Protein Misfolding