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GDF15 attenuates sepsis-induced myocardial dysfunction by inhibiting cardiomyocytes ferroptosis via the SOCS1/GPX4 signaling pathway

Xiayun Li, He Sun, Liyun Zhang, Hongliang Liang, Bin Zhang, Jiachang Yang, Xiangyan Peng, Jingwei Sun, Yang Zhou, Mengen Zhai, Liqing Jiang, Hanzhao Zhu, Weixun Duan

2024European Journal of Pharmacology31 citationsDOI

Topics & Concepts

GPX4GDF15SepsisOxidative stressInflammationLipid peroxidationReactive oxygen speciesMedicineProgrammed cell deathCancer researchImmunologyPharmacologyInternal medicineCell biologyBiologyGlutathione peroxidaseSuperoxide dismutaseApoptosisBiochemistryGDF15 and Related BiomarkersMacrophage Migration Inhibitory FactorCircular RNAs in diseases
GDF15 attenuates sepsis-induced myocardial dysfunction by inhibiting cardiomyocytes ferroptosis via the SOCS1/GPX4 signaling pathway | Litcius