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TRPC channel-derived calcium fluxes differentially regulate ATP and flow-induced activation of eNOS

Tenderano T. Muzorewa, Donald G. Buerk, Dov Jaron, Steven M. Kurtz

2021Nitric Oxide11 citationsDOIOpen Access PDF

Topics & Concepts

TRPCEnosTRPC3Transient receptor potential channelORAI1ChemistryNitric oxideVasodilationCell biologyTRPC1Nitric Oxide Synthase Type IIIStimulationCalciumCalcium channelVoltage-dependent calcium channelInternal medicineNitric oxide synthaseEndocrinologyBiologyBiochemistryReceptorMedicineOrganic chemistryIon Channels and ReceptorsCardiovascular, Neuropeptides, and Oxidative Stress ResearchNeurobiology and Insect Physiology Research
TRPC channel-derived calcium fluxes differentially regulate ATP and flow-induced activation of eNOS | Litcius