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Experimental autoimmune encephalopathy (EAE)-induced hippocampal neuroinflammation and memory deficits are prevented with the non-opioid TLR2/TLR4 antagonist (+)-naltrexone

Andrew J. Kwilasz, Laurel S. Todd, Julissa Chante Duran-Malle, Anouk E.W. Schrama, Eric H. Mitten, Tracey A. Larson, Madison A. Clements, Kevin M. Harris, Scott T. Litwiler, Xiaohui Wang, Anne‐Marie van Dam, Steven F. Maier, Kenner C. Rice, Linda R. Watkins, Ruth M. Barrientos

2020Behavioural Brain Research25 citationsDOIOpen Access PDF

Topics & Concepts

NeuroinflammationHippocampusNeuroscienceHippocampal formationTLR4PsychologyMedicineExperimental autoimmune encephalomyelitisProinflammatory cytokineInflammationImmunologyCentral nervous systemNeuroinflammation and Neurodegeneration MechanismsImmune Response and InflammationMultiple Sclerosis Research Studies
Experimental autoimmune encephalopathy (EAE)-induced hippocampal neuroinflammation and memory deficits are prevented with the non-opioid TLR2/TLR4 antagonist (+)-naltrexone | Litcius