Experimental autoimmune encephalopathy (EAE)-induced hippocampal neuroinflammation and memory deficits are prevented with the non-opioid TLR2/TLR4 antagonist (+)-naltrexone
Andrew J. Kwilasz, Laurel S. Todd, Julissa Chante Duran-Malle, Anouk E.W. Schrama, Eric H. Mitten, Tracey A. Larson, Madison A. Clements, Kevin M. Harris, Scott T. Litwiler, Xiaohui Wang, Anne‐Marie van Dam, Steven F. Maier, Kenner C. Rice, Linda R. Watkins, Ruth M. Barrientos
Topics & Concepts
NeuroinflammationHippocampusNeuroscienceHippocampal formationTLR4PsychologyMedicineExperimental autoimmune encephalomyelitisProinflammatory cytokineInflammationImmunologyCentral nervous systemNeuroinflammation and Neurodegeneration MechanismsImmune Response and InflammationMultiple Sclerosis Research Studies