Reduction of ADAMTS13 Levels Predicts Mortality in SARS-CoV-2 Patients
Giovanni Luca Tiscia, Giovanni Favuzzi, Antonio De Laurenzo, Filomena Cappucci, Lucia Fischetti, Lazzaro Di Mauro, Giuseppe Miscio, Antonio Mirijello, Elena Chinni, Elvira Grandone, on behalf of CSS COVID-19 Group
Abstract
In SARS-CoV-2 (severe acute respiratory syndrome coronavirus 2) infected patients, hemostasis shows an uncontrolled activation, with a high D-dimer concentration.[1] [2] It has been also shown that the SARS-CoV-2 can promote endotheliitis,[3] and in turn, pulmonary endothelial damage fostering thrombogenesis.[4]
Topics & Concepts
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2)Coronavirus disease 2019 (COVID-19)ADAMTS13D-dimer2019-20 coronavirus outbreakCoronavirusMedicineHemostasisBetacoronavirusSevere acute respiratory syndromeCardiologyInternal medicinePandemicVon Willebrand factorVirologyDiseaseInfectious disease (medical specialty)OutbreakPlateletComplement system in diseasesCOVID-19 Clinical Research StudiesSARS-CoV-2 and COVID-19 Research