Maslinic acid differentially exploits the MAPK pathway in estrogen-positive and triple-negative breast cancer to induce mitochondrion-mediated, caspase-independent apoptosis
Ritu Jain, Abhinav Grover
Topics & Concepts
ApoptosisTriple-negative breast cancerCancer researchBreast cancerEstrogen receptorProgrammed cell deathEstrogenCancerBiologyCell growthMAPK/ERK pathwayInternal medicineChemistryEndocrinologyMedicineSignal transductionCell biologyBiochemistryNatural product bioactivities and synthesisCancer, Lipids, and MetabolismTannin, Tannase and Anticancer Activities