A recurrent chromosomal inversion suffices for driving escape from oncogene-induced senescence via subTAD reorganization
Christos Zampetidis, Panagiotis Galanos, Andriani Angelopoulou, Yajie Zhu, Aikaterini Polyzou, Timokratis Karamitros, Athanassios Kotsinas, Nefeli Lаgopati, Ioanna Mourkioti, Reza Mirzazadeh, A. Polyzos, Silvano Garnerone, Athanasia Mizi, Eduardo Gade Gusmao, Konstantinos Sofiadis, Zita Gál, Dorthe Helena Larsen, Dafni‐Eleftheria Pefani, Marco Demaria, Aristotelis Tsirigos, Nicola Crosetto, Apolinar Maya‐Mendoza, Angelos Papaspyropoulos, Konstantinos Evangelou, Jiří Bártek, Argyris Papantonis, Vassilis G. Gorgoulis
Topics & Concepts
BiologySenescencePhenotypeOncogeneCell biologySuppressorGenome instabilityTranscription factorEctopic expressionGeneCell cycleGeneticsCancer researchDNA damageDNATelomeres, Telomerase, and SenescenceGenomics and Chromatin DynamicsEpigenetics and DNA Methylation