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Elevated Cytokine Levels in Plasma of Patients with SARS-CoV-2 Do Not Contribute to Pulmonary Microvascular Endothelial Permeability

Anita Kovács‐Kása, Abdelrahman A. Zaied, Silvia Leanhart, Murat Koseoglu, Supriya Sridhar, Rudolf Lucas, David Fulton, José A. Vázquez, Brian H. Annex

2022Microbiology Spectrum17 citationsDOIOpen Access PDF

Abstract

. Known endothelial barrier-disruptive agents and proposed mediators of increased endothelial permeability in SARS-CoV-2, induced changes in permeability that were smaller in magnitude and shorter in duration than plasma from patients with SARS-CoV-2. The effect on endothelial cell permeability of plasma from patients with SARS-CoV-2 was heat-labile. The main plasma factor that causes the increased endothelial permeability remains to be identified. Our study provides a possible approach for future studies to understand the underlying mechanisms leading to vascular injury in SARS-CoV-2 infections.

Topics & Concepts

CytokineVascular permeabilityVascular endothelial growth factorMedicineTumor necrosis factor alphaEndothelial stem cellImmunologyThrombinAntibodyInflammationEndotheliumEndothelial activationInternal medicineBiologyPlateletIn vitroVEGF receptorsBiochemistryCOVID-19 Clinical Research StudiesLong-Term Effects of COVID-19SARS-CoV-2 and COVID-19 Research
Elevated Cytokine Levels in Plasma of Patients with SARS-CoV-2 Do Not Contribute to Pulmonary Microvascular Endothelial Permeability | Litcius