Molecular mechanisms by which iNOS uncoupling can induce cardiovascular dysfunction during sepsis: Role of posttranslational modifications (PTMs)
Nivin Sharawy, Christine Lehmann
Topics & Concepts
SepsisNitric oxide synthasePathogenesisPathologicalNitric oxideMechanism (biology)Gene isoformHomeostasisBioinformaticsBiologyMedicineNeuroscienceImmunologyCell biologyBiochemistryInternal medicineGenePhilosophyEpistemologyNitric Oxide and Endothelin EffectsAnalytical Chemistry and SensorsNeutrophil, Myeloperoxidase and Oxidative Mechanisms