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ITGA3 promotes pancreatic cancer progression through HIF1α- and c-Myc-driven glycolysis in a collagen I-dependent autocrine manner

Rong-Kun Li, Min Ji, Shengqiao Fu, Jichun Gu, Dejun Liu, Lu Wang, Xiao Yuan, Yi Wen, Chunhua Dai, H Li

2024Cancer Gene Therapy22 citationsDOI

Topics & Concepts

Pancreatic cancerAutocrine signallingCancer researchPI3K/AKT/mTOR pathwayCancerCancer cellTumor progressionDesmoplasiaFocal adhesionProtein kinase BMetastasisChemistryInternal medicineBiologySignal transductionCell biologyMedicineReceptorPancreatic and Hepatic Oncology ResearchCancer Cells and MetastasisCancer, Hypoxia, and Metabolism
ITGA3 promotes pancreatic cancer progression through HIF1α- and c-Myc-driven glycolysis in a collagen I-dependent autocrine manner | Litcius