Litcius/Paper detail

Glomerulonephritis Caused by <i>Bartonella</i> spp. Infective Endocarditis: The Difficulty and Importance of Differentiation from Anti-neutrophil Cytoplasmic Antibody-related Rapidly Progressive Glomerulonephritis

Ayumi Yoshifuji, Yuuka Hibino, Motoaki Komatsu, Seiichi Yasuda, Koji Hosoya, Emi Kobayashi, Yuko Baba, Shigemichi Hirose, Akinori Hashiguchi, Yoshihiko Kanno, Munekazu Ryuzaki

2021Internal Medicine11 citationsDOIOpen Access PDF

Abstract

A 65-year-old man with valvular disorder presented to his physician because of widespread purpura in both lower extremities. Blood tests showed elevated serum creatinine levels and proteinase 3-anti-neutrophil cytoplasmic antibody (ANCA) with hematuria, suggesting ANCA-related rapidly progressive glomerulonephritis (RPGN). Although multiple blood cultures were negative, transthoracic echocardiography revealed warts in the valves, and a renal biopsy also showed findings of glomerular infiltration by mononuclear leukocytes and C3 deposition in the glomeruli, suggesting infection-related glomerulonephritis. Later, Bartonella antibody turned positive. Antimicrobial treatment improved the purpura and renal function without any recurrence. ANCA-positive RPGN requires the exclusion of infective endocarditis, especially that induced by Bartonella spp.

Topics & Concepts

MedicineRapidly progressive glomerulonephritisGlomerulonephritisInfective endocarditisRenal biopsyBartonellaPathologyRenal functionEndocarditisVasculitisAntibodyPurpura (gastropod)ImmunologyBiopsyInternal medicineKidneyVirologyDiseaseBiologyEcologyBartonella species infections researchInfective Endocarditis Diagnosis and ManagementStreptococcal Infections and Treatments