Absence of Receptor for Advanced Glycation End Product (RAGE) Reduces Inflammation and Extends Survival in the hSOD1G93A Mouse Model of Amyotrophic Lateral Sclerosis
John D. Lee, Tanya S. McDonald, Jenny N. Fung, Trent M. Woodruff
Topics & Concepts
Amyotrophic lateral sclerosisNeuroinflammationNeurodegenerationRage (emotion)MedicineSOD1Advanced glycation end-productNeuroscienceSpinal cord injuryDenervationInflammationSpinal cordImmunologyGlycationReceptorBiologyPathologyInternal medicineDiseaseAmyotrophic Lateral Sclerosis ResearchNeuroinflammation and Neurodegeneration Mechanisms