Diannexin alleviates myocardial ischemia–reperfusion injury by orchestrating cardiomyocyte oxidative damage, macrophage polarization and fibrotic process by TLR4-NF-kB-mediated inactivation of NLRP3 inflammasome
Lin Zhang, Songlin Zhao, Yaqi Wang
Topics & Concepts
InflammasomeApoptosisReperfusion injuryMacrophage polarizationCell biologyOxidative stressTLR4Reactive oxygen speciesSignal transductionChemistryInflammationMacrophageBiologyPharmacologyCancer researchMedicineIschemiaImmunologyInternal medicineEndocrinologyBiochemistryIn vitroCardiac Ischemia and ReperfusionS100 Proteins and AnnexinsCardiac Fibrosis and Remodeling