Tenascin-C Induces Phenotypic Changes in Fibroblasts to Myofibroblasts with High Contractility through the Integrin αvβ1/Transforming Growth Factor β/SMAD Signaling Axis in Human Breast Cancer
Daisuke Katoh, Yuji Kozuka, Aya Noro, Tomoko Ogawa, Kyoko Imanaka‐Yoshida, Toshimichi Yoshida
Topics & Concepts
SMADMyofibroblastTransforming growth factorTenascinCancer researchPhenotypeContractilityTenascin CSignal transductionCell biologyIntegrinBreast cancerBiologyFibronectinsTransforming growth factor betaPathologyCancerEndocrinologyMedicineInternal medicineFibronectinFibrosisExtracellular matrixBiochemistryCellGeneCell Adhesion Molecules ResearchCellular Mechanics and InteractionsCancer Cells and Metastasis