Litcius/Paper detail

<i>In Situ</i> Coassembly Induced Mitochondrial Aggregation Activated Drug-Resistant Tumor Treatment

Xianyin Dai, Bing Zhang, Qilin Yu, Yu Liu

2022Journal of Medicinal Chemistry23 citationsDOI

Abstract

Macrocyclic supramolecular coassembly is the current research hotspot for tumor treatment. Herein, we report a multivalent supramolecular coassembly strategy, which not only acquires long-time phosphorescent labeling of mitochondrial aggregation but also strongly enhances chemotherapeutic efficiency against drug-resistant tumors. The mitochondrial aggregation depends on cucurbit[8]uril-mediated cross-linkage of the hyaluronic acid polymer grafted by 4-bromophenylpyridium and mitochondrion-targeting peptide (HABMitP) residing on the mitochondria, taking advantage of the 2:1 homoternary host–guest complexation between cucurbit[8]uril and 4-bromophenylpyridium with an extraordinary binding constant (6.24 × 1012 M–2). In cisplatin-resistant MCF-7 tumor cells, the assembly induced mitochondrial aggregation substantially enhances the antitumor efficiency of cisplatin, with the ratio of apoptotic cells increasing from 43% to 96% compared to treatment with cisplatin alone, and thoroughly inhibits tumor growth in vivo. This study provides a novel way for biological phosphorescent imaging and treatment of drug-resistant cancers.

Topics & Concepts

ChemistryCisplatinSupramolecular chemistryIn vivoMitochondrionPhosphorescenceIn vitroHyaluronic acidBiophysicsPeptideDrugApoptosisBiochemistryCombinatorial chemistryCell biologyCancer researchPharmacologyChemotherapyBiologyMoleculeOrganic chemistryAnatomyGeneticsBiotechnologyFluorescencePhysicsQuantum mechanicsSupramolecular Chemistry and ComplexesSupramolecular Self-Assembly in MaterialsBoron Compounds in Chemistry