Litcius/Paper detail

Discovery of Highly Potent and BMPR2-Selective Kinase Inhibitors Using DNA-Encoded Chemical Library Screening

Ram K. Modukuri, Diana Monsivais, Feng Li, Murugesan Palaniappan, Kurt M. Bohren, Zhi Tan, Angela F. Ku, Yong Wang, Chandrashekhar Madasu, Jianyuan Li, Suni Tang, Gabriella Miklóssy, Stephen Palmer, Damian W. Young, Martin M. Matzuk

2023Journal of Medicinal Chemistry29 citationsDOIOpen Access PDF

Abstract

The discovery of monokinase-selective inhibitors for patients is challenging because the 500+ kinases encoded by the human genome share highly conserved catalytic domains. Until now, no selective inhibitors unique for a single transforming growth factor β (TGFβ) family transmembrane receptor kinase, including bone morphogenetic protein receptor type 2 (BMPR2), have been reported. This dearth of receptor-specific kinase inhibitors hinders therapeutic options for skeletal defects and cancer as a result of an overactivated BMP signaling pathway. By screening 4.17 billion “unbiased” and “kinase-biased” DNA-encoded chemical library molecules, we identified hits CDD-1115 and CDD-1431, respectively, that were low-nanomolar selective kinase inhibitors of BMPR2. Structure–activity relationship studies addressed metabolic lability and high-molecular-weight issues, resulting in potent and BMPR2-selective inhibitor analogs CDD-1281 (IC 50 = 1.2 nM) and CDD-1653 (IC 50 = 2.8 nM), respectively. Our work demonstrates that DNA-encoded chemistry technology (DEC-Tec) is reliable for identifying novel first-in-class, highly potent, and selective kinase inhibitors.

Topics & Concepts

BMPR2KinaseChemistryBiochemistryDrug discoveryTransmembrane proteinActivin receptorSmall moleculeBone morphogenetic proteinReceptorGeneTGF-β signaling in diseasesHeterotopic Ossification and Related ConditionsCancer, Hypoxia, and Metabolism