HMOX1 silencing prevents doxorubicin-induced cardiomyocyte injury, mitochondrial dysfunction, and ferroptosis by downregulating CTGF
Jia Ming Qian, Wenting Wan, Min Fan
Topics & Concepts
CTGFHMOX1ApoptosisViability assayGene silencingCell biologyProgrammed cell deathMitochondrionChemistryDoxorubicinMolecular biologyCancer researchBiologyGrowth factorBiochemistryGeneticsChemotherapyReceptorEnzymeHemeGeneHeme oxygenaseFerroptosis and cancer prognosisConnective Tissue Growth Factor ResearchGDF15 and Related Biomarkers