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Monocyte and bone marrow macrophage transcriptional phenotypes in systemic juvenile idiopathic arthritis reveal TRIM8 as a mediator of IFN-γ hyper-responsiveness and risk for macrophage activation syndrome

Grant S. Schulert, Alex Pickering, Thuy Do, Sanjeev Dhakal, Ndate Fall, Daniel Schnell, Mario Medvedovic, Nathan Salomonis, Sherry Thornton, Alexei A. Grom

2020Annals of the Rheumatic Diseases50 citationsDOI

Topics & Concepts

MonocyteGene knockdownImmunologyMacrophageMedicineMacrophage activation syndromeTranscriptomeCytokineChemokineDownregulation and upregulationArthritisBone marrowCD16InterferonPhenotypeBiologyCell biologyInflammationIn vitroImmune systemCell cultureGene expressionGeneGeneticsCD8CD3Autoimmune and Inflammatory Disorders ResearchImmune cells in cancerinterferon and immune responses
Monocyte and bone marrow macrophage transcriptional phenotypes in systemic juvenile idiopathic arthritis reveal TRIM8 as a mediator of IFN-γ hyper-responsiveness and risk for macrophage activation syndrome | Litcius