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Roles of ERK/Akt signals in mitochondria-dependent and endoplasmic reticulum stress-triggered neuronal cell apoptosis induced by 4-methyl-2,4-bis(4-hydroxyphenyl)pent-1-ene, a major active metabolite of bisphenol A

Chun-Fa Huang, Shing‐Hwa Liu, Chin‐Chuan Su, Kai‐Min Fang, Cheng-Chieh Yen, Ching‐Yao Yang, Feng‐Cheng Tang, Jui‐Ming Liu, Chin-Ching Wu, Kuan-I Lee, Ya‐Wen Chen

2021Toxicology39 citationsDOI

Topics & Concepts

Unfolded protein responseATF6Endoplasmic reticulumCell biologyProtein kinase AProgrammed cell deathApoptosisKinasep38 mitogen-activated protein kinasesBiologySignal transductionProtein kinase BChemistryBiochemistryEffects and risks of endocrine disrupting chemicalsCarcinogens and Genotoxicity AssessmentNuclear Receptors and Signaling
Roles of ERK/Akt signals in mitochondria-dependent and endoplasmic reticulum stress-triggered neuronal cell apoptosis induced by 4-methyl-2,4-bis(4-hydroxyphenyl)pent-1-ene, a major active metabolite of bisphenol A | Litcius