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Taxifolin and Sorghum Ethanol Extract Protect against Hepatic Insulin Resistance via the miR-195/IRS1/PI3K/AKT and AMPK Signalling Pathways

Hana Lee, Won Tae Jeong, Yoon‐Sup So, Heung‐Bin Lim, Junsoo Lee

2021Antioxidants25 citationsDOIOpen Access PDF

Abstract

This study aimed to evaluate the effects of taxifolin and sorghum ethanol extract on free fatty acid (FFA)-induced hepatic insulin resistance. FFA treatment decreased glucose uptake by 16.2% compared with that in the control, whereas taxifolin and sorghum ethanol extract increased the glucose uptake. Additionally, taxifolin and sorghum ethanol extract increased the expression of p-PI3K, p-IRS1, p-AKT, p-AMPK, and p-ACC in FFA-induced hepatocytes. Furthermore, FFA treatment increased the expression of miR-195. However, compared with the FFA treatment, treatment with taxifolin and sorghum ethanol extract decreased miR-195 expression in a dose-dependent manner. Taxifolin and sorghum ethanol extract enhanced p-IRS1, p-PI3K, p-AMPK, p-AKT, and p-ACC expression by suppressing miR-195 levels in miR-195 mimic- or inhibitor-transfected cells. These results indicate that taxifolin and sorghum ethanol extract attenuate insulin resistance by regulating miR-195 expression, which suggests that taxifolin and sorghum ethanol extract may be useful antidiabetic agents.

Topics & Concepts

TaxifolinIRS1AMPKPI3K/AKT/mTOR pathwayChemistryKaempferolInsulin resistanceProtein kinase BSorghumPharmacologyInsulinBiochemistryQuercetinInsulin receptorSignal transductionEndocrinologyBiologyProtein kinase AKinaseAntioxidantEcologyMicroRNA in disease regulationCancer-related molecular mechanisms researchRNA modifications and cancer
Taxifolin and Sorghum Ethanol Extract Protect against Hepatic Insulin Resistance via the miR-195/IRS1/PI3K/AKT and AMPK Signalling Pathways | Litcius