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The lack of functional nicotinamide nucleotide transhydrogenase only moderately contributes to the impairment of glucose tolerance and glucose-stimulated insulin secretion in C57BL/6J vs C57BL/6N mice

Anne-Françoise Close, Heeyoung Chae, Jean‐Christophe Jonas

2021Diabetologia21 citationsDOIOpen Access PDF

Topics & Concepts

Internal medicineEndocrinologyBiologyNAD+ kinaseMitochondrionImpaired glucose toleranceGlucose homeostasisInsulinBiochemistryInsulin resistanceMedicineEnzymeATP Synthase and ATPases ResearchMitochondrial Function and PathologyAdipose Tissue and Metabolism
The lack of functional nicotinamide nucleotide transhydrogenase only moderately contributes to the impairment of glucose tolerance and glucose-stimulated insulin secretion in C57BL/6J vs C57BL/6N mice | Litcius