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TP53-induced glycolysis and apoptosis regulator (TIGAR) ameliorates lysosomal damage in the 1-methyl-4-phenyl-1, 2, 3, 6-tetrahydropyridine-mediated mouse model of Parkinson’s disease

Jianbin Ge, Hongyan Lin, Jie Yang, QiQi Li, Jing-si Zhou, Zheng‐Hong Qin, Feng Wu

2020Toxicology Letters26 citationsDOI

Topics & Concepts

Substantia nigraPars compactaMPTPNeuroprotectionOxidative stressNeurotoxinCell biologyReactive oxygen speciesDownregulation and upregulationChemistryLysosomeAutophagyBiologyApoptosisDopaminergicBiochemistryDopaminePharmacologyNeuroscienceEnzymeGeneParkinson's Disease Mechanisms and TreatmentsAutophagy in Disease and TherapyCalcium signaling and nucleotide metabolism
TP53-induced glycolysis and apoptosis regulator (TIGAR) ameliorates lysosomal damage in the 1-methyl-4-phenyl-1, 2, 3, 6-tetrahydropyridine-mediated mouse model of Parkinson’s disease | Litcius