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Structural Basis for the Selective Inhibition of HDAC10, the Cytosolic Polyamine Deacetylase

Corey J. Herbst‐Gervasoni, Raphael R. Steimbach, Michael Morgen, Aubry K. Miller, David W. Christianson

2020ACS Chemical Biology41 citationsDOIOpen Access PDF

Abstract

helix containing the P(E,A)CE motif that sterically constricts the active site and an electrostatic "gatekeeper," E274, that confers selectivity for cationic polyamine substrates. To accelerate drug design efforts targeting human HDAC10, we now report the preparation of "humanized" zebrafish HDAC10 in which two amino acid substitutions, A24E and D94A, yield an active site contour more similar to that of human HDAC10. X-ray crystal structures of this HDAC10 variant complexed with Tubastatin A and indole analogues bearing pendant tertiary amines reveal that inhibitors capable of hydrogen bonding with gatekeeper E274 exhibit high affinity and selectivity for HDAC10 over HDAC6 (the other class IIb isozyme). Moreover, these structures reveal that the P(E,A)CE motif helix can shift by up to 2 Å to accommodate the binding of bulky inhibitors. Thus, slender polyamine-like inhibitor structures are not exclusively required for selective, high affinity binding to HDAC10. Indeed, the flexibility of the P(E,A)CE motif helix could conceivably enable the binding of certain protein substrates.

Topics & Concepts

HDAC10ChemistryHistone deacetylaseBiochemistryCancer researchBiologyHistoneGeneHistone Deacetylase Inhibitors ResearchEpigenetics and DNA MethylationAdenosine and Purinergic Signaling
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