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Kallistatin/Serpina3c inhibits cardiac fibrosis after myocardial infarction by regulating glycolysis via Nr4a1 activation

Jingjing Ji, Ling-lin Qian, Yi Zhu, Yu Jiang, Jiaqi Guo, Ya Wu, Ziwei Yang, Yuyu Yao, Genshan Ma

2022Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease45 citationsDOI

Topics & Concepts

GlycolysisFibrosisHeart failureMyocardial infarctionCardiac fibrosisVentricular remodelingMyocardial fibrosisMedicineInternal medicineCancer researchAcetylationTranscriptomeEndocrinologyGene expressionCardiologyPharmacologyChemistryGeneMetabolismBiochemistryNuclear Receptors and SignalingCardiac Fibrosis and RemodelingNerve injury and regeneration
Kallistatin/Serpina3c inhibits cardiac fibrosis after myocardial infarction by regulating glycolysis via Nr4a1 activation | Litcius