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Strong influenza-induced T <sub>FH</sub> generation requires CD4 effectors to recognize antigen locally and receive signals from continuing infection

Priyadharshini Devarajan, Allen M. Vong, Catherine H. Castonguay, Olivia Kugler‐Umana, Bianca Bautista, Michael C. Jones, Karen A. Kelly, Jingya Xia, Susan L. Swain

2022Proceedings of the National Academy of Sciences21 citationsDOIOpen Access PDF

Abstract

Significance Influenza infection elicits strong, long-lived protective antibodies, but most current influenza vaccines give weaker, short-lived protection. We noted that live virus is still replicating, making antigen and causing inflammation at 7 d postinfection (dpi), while an inactivated vaccine provides antigen for at most 4 dpi. We show that the generation of key T follicular helper cells (T FH ) requires they recognize antigen locally at 6 dpi in the presence of ongoing viral infection. This creates a checkpoint that restricts T FH responses to dangerous infections that persist through the checkpoint. Using a live attenuated vaccine, akin to Flumist, we found that adding a second dose at 6 d generated a strong T FH response, suggesting an approach to improve vaccine strategies.

Topics & Concepts

AntigenVirologyImmunologyBiologyInfluenza vaccineEffectorAntibodyVaccinationMedicineImmune Cell Function and InteractionT-cell and B-cell ImmunologyImmunotherapy and Immune Responses
Strong influenza-induced T <sub>FH</sub> generation requires CD4 effectors to recognize antigen locally and receive signals from continuing infection | Litcius