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Fis Contributes to Resistance of Pseudomonas aeruginosa to Ciprofloxacin by Regulating Pyocin Synthesis

Yuqing Long, Weixin Fu, Su Wang, Xuan Deng, Yongxin Jin, Fang Bai, Zhihui Cheng, Weihui Wu

2020Journal of Bacteriology20 citationsDOIOpen Access PDF

Abstract

Pseudomonas aeruginosa is an important opportunistic pathogenic bacterium that causes various acute and chronic infections in human, especially in patients with compromised immunity, cystic fibrosis (CF), and/or severe burn wounds. About 60% of cystic fibrosis patients have a chronic respiratory infection caused by P. aeruginosa . The bacterium is intrinsically highly resistant to antibiotics, which greatly increases difficulties in clinical treatment. Therefore, it is critical to understand the mechanisms and the regulatory pathways that are involved in antibiotic resistance. In this study, we elucidated a novel regulatory pathway that controls the bacterial resistance to fluoroquinolone antibiotics, which enhances our understanding of how P. aeruginosa responds to ciprofloxacin.

Topics & Concepts

Pseudomonas aeruginosaCiprofloxacinCystic fibrosisBiologyMicrobiologyAntibioticsAntibiotic resistanceBacteriaImmunologyGeneticsAntibiotic Resistance in BacteriaBacterial biofilms and quorum sensingCystic Fibrosis Research Advances
Fis Contributes to Resistance of Pseudomonas aeruginosa to Ciprofloxacin by Regulating Pyocin Synthesis | Litcius