CD4+ T cells reactive to a hybrid peptide from insulin-chromogranin A adopt a distinct effector fate and are pathogenic in autoimmune diabetes
Jason S. Mitchell, Justin A. Spanier, Alexander J. Dwyer, Todd P. Knutson, Mohannad Alkhatib, Gina Qian, Matthew E. Weno, Yixin Chen, Zachary R. Shaheen, Christopher Tucker, Takashi Kangas, Milagros Silva Morales, Nubia Silva, Tsuneyasu Kaisho, Michael A. Farrar, Brian T. Fife
Topics & Concepts
EffectorBiologyIsletChromogranin ADiabetes mellitusAutoimmune diabetesAutoimmunityAutoimmune diseaseInsulinPathogenicityImmunologyEndocrinologyCell biologyImmune systemAntibodyMicrobiologyImmunohistochemistryDiabetes and associated disordersPancreatic function and diabetesDiabetes Management and Research