Pathogen-induced tissue-resident memory T <sub>H</sub> 17 (T <sub>RM</sub> 17) cells amplify autoimmune kidney disease
Christian F. Krebs, Daniel Reimers, Yu Zhao, Hans‐Joachim Paust, Patricia Bartsch, Sarah Núñez, Mariana V. Rosemblatt, Malte Hellmig, Christoph Kilian, Alina Borchers, Leon Enk, Michael Zinke, Martina Becker, Joanna Schmid, Stefanie Klinge, Milagros N. Wong, Victor G. Puelles, Constantin Schmidt, Tabea Bertram, Natascha E. Stumpf, Elion Hoxha, Catherine Meyer‐Schwesinger, Maja T. Lindenmeyer, Clemens D. Cohen, Michael Rink, Christian Kurts, Sören Franzenburg, Friedrich Koch‐Nolte, Jan‐Eric Turner, Jan-Hendrik Riedel, Samuel Huber, Nicola Gagliani, Tobias B. Huber, Thorsten Wiech, Holger Rohde, Marı́a Rosa Bono, Stefan Bonn, Ulf Panzer, Hans‐Willi Mittrücker
Abstract
CD4 + T RM 17 cells become activated by inflammatory cytokines to produce IL-17A and thereby promote autoimmunity.