Litcius/Paper detail

Degradation of the NOTCH intracellular domain by elevated autophagy in osteoblasts promotes osteoblast differentiation and alleviates osteoporosis

Gota Yoshida, Tsuyoshi Kawabata, Hyota Takamatsu, Shotaro Saita, Shuhei Nakamura, Keizo Nishikawa, Mari Fujiwara, Yusuke Enokidani, Tadashi Yamamuro, Keisuke Tabata, Maho Hamasaki, Masaru Ishii, Atsushi Kumanogoh, Tamotsu Yoshimori

2022Autophagy86 citationsDOIOpen Access PDF

Abstract

: ALPL: alkaline phosphatase, liver/bone/kidney; BCIP/NBT: 5-bromo-4-chloro-3'-indolyl phosphate/nitro blue tetrazolium; BMD: bone mineral density; BV/TV: bone volume/total bone volume; MAP1LC3/LC3: microtubule-associated protein 1 light chain 3; MTOR: mechanistic target of rapamycin kinase; NICD: NOTCH intracellular domain; RB1CC1/FIP200: RB1-inducible coiled-coil 1; RUBCN/Rubicon: RUN domain and cysteine-rich domain containing, Beclin 1-interacting protein; SERM: selective estrogen receptor modulator; TNFRSF11B/OCIF: tumor necrosis factor receptor superfamily, member 11b (osteoprotegerin).

Topics & Concepts

AutophagyOsteoblastOsteoclastCell biologyDownregulation and upregulationNotch signaling pathwayIntracellularOsteoporosisChemistryRUNX2BiologyRegulatorBone remodelingBone resorptionTranscription factorInternal medicineEndocrinologyPhenotypeCancer researchBone mineralBone morphogenetic protein 2Cellular differentiationRegulation of gene expressionBone cellBone Metabolism and DiseasesAutophagy in Disease and TherapyBone health and osteoporosis research