Litcius/Paper detail

Treatment with a triazole inhibitor of the mitochondrial permeability transition pore fully corrects the pathology of sapje zebrafish lacking dystrophin

A. Stocco, Natalia Smolina, Patrizia Sabatelli, Justina Šileikytė, Edoardo Artusi, Vincent Mouly, Michael S. Cohen, Michael Forte, Marco Schiavone, Paolo Bernardi

2021Pharmacological Research36 citationsDOI

Topics & Concepts

ZebrafishDystrophinMitochondrial permeability transition poreMyogenesisDuchenne muscular dystrophyIn vivoMitochondrionCell biologyBiologyMyocyteChemistryPharmacologyApoptosisBiochemistryProgrammed cell deathGeneticsGeneMuscle Physiology and DisordersAdipose Tissue and MetabolismMitochondrial Function and Pathology
Treatment with a triazole inhibitor of the mitochondrial permeability transition pore fully corrects the pathology of sapje zebrafish lacking dystrophin | Litcius