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The microglial NLRP3 inflammasome is involved in human SARS-CoV-2 cerebral pathogenicity: A report of three post-mortem cases

Viviana Falcón Cama, Javier Marín‐Prida, Nelson Acosta‐Rivero, Emilio F. Acosta, Leonardo Oramas Díaz, Ana Victoria Casadesús, Briandy Fernández-Marrero, Nathalie Gilva-Rodríguez, Daina Cremata-García, Majel Cervantes‐Llanos, Beatriz Piniella‐Matamoros, Daphne E Sanchez, Leticia del Rosario-Cruz, Israel Borrajero, Angelina Díaz García, Yorexis González, Eduardo Pentón‐Arias, Teresita Montero-González, Gerardo Guillén, Giselle Pentón‐Rol

2021Journal of Neuroimmunology48 citationsDOIOpen Access PDF

Topics & Concepts

ColocalizationInflammasomeMicrogliaSevere acute respiratory syndrome coronavirus 2 (SARS-CoV-2)BiologyNeuroinflammationInflammationImmunofluorescenceVirologyCentral nervous systemPathologyCoronavirus disease 2019 (COVID-19)PathogenicityImmunologyMedicineNeuroscienceAntibodyMicrobiologyInfectious disease (medical specialty)DiseaseLong-Term Effects of COVID-19COVID-19 Clinical Research StudiesNeuroinflammation and Neurodegeneration Mechanisms
The microglial NLRP3 inflammasome is involved in human SARS-CoV-2 cerebral pathogenicity: A report of three post-mortem cases | Litcius