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Intranasal 15d-PGJ2 ameliorates brain glucose hypometabolism via PPARγ-dependent activation of PGC-1α/GLUT4 signalling in APP/PS1 transgenic mice

Zongyang Li, Yuan Zhang, Yue‐yang Zheng, Wenlan Liu, Xiejun Zhang, Weiping Li, Di Zhang, Qian Cai, Sicen Wang, Xiangbao Meng, Guodong Huang

2021Neuropharmacology18 citationsDOI

Topics & Concepts

NeuroinflammationGLUT4Genetically modified mouseAgonistEndocrinologyPeroxisome proliferator-activated receptorPharmacologyHippocampusInternal medicineGlucose uptakePertussis toxinDownregulation and upregulationGlucose transporterChemistryTransgeneReceptorBiologyMedicineInsulinInflammationBiochemistryG proteinGenePeroxisome Proliferator-Activated ReceptorsInflammatory mediators and NSAID effectsEicosanoids and Hypertension Pharmacology
Intranasal 15d-PGJ2 ameliorates brain glucose hypometabolism via PPARγ-dependent activation of PGC-1α/GLUT4 signalling in APP/PS1 transgenic mice | Litcius